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Corrigendum for Cotero and Routh, Am J Physiol Endocrinol Metab 296 (5) E1101-E1109.
Am J Physiol Endocrinol Metab 297: E560-E561, 2009; doi:10.1152/ajpendo.zh1-5738-corr.2009
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CORRIGENDUM

Corrigendum

Volume 296, May 2009

Volume 59, May 2009

Pages E1101–E1109: Cotero VE, Routh VH. Insulin blunts the response of glucose-excited neurons in the ventrolateral-ventromedial hypothalamic nucleus to decreased glucose; doi: 10.1152/ajpendo.90932.2008; http://ajpendo.physiology.org/cgi/content/full/296/05/E1101.

Subsequent to publication of this article, the authors discovered the following two errors.

In Fig. 4, the legends for the lines were accidentally inverted.


Figure 4
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  		Fig. 4. Current-voltage relationships indicate that the insulin-sensitive conductance reverses near the theoretical K+ equilibrium potential (–99 mV) in 5 (A), 2.5 (B), and 0.1 mM glucose (C).

 
In Fig. 5, a decimal error was made in two of the data points during data transfer for generation of the final version of the figure; thus, the reported percent change in input resistance for insulin in 0.1 mM glucose was incorrectly elevated. That average percent change has now been corrected and is consistent with the values in Fig. 2C. The corrected figures with legends are reproduced below.


Figure 5
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  		Fig. 5. Tolbutamide (T; 100 µM) blocks the effects of insulin on the input resistance (IR) of VL-VMN GE neurons in 5 (A), 2.5 (B), and 0.1 mM glucose (C). D: current-voltage relationship for the effect of insulin in 0.1 mM glucose in the present and absence of tolbutamide reverses near the theoretical K+equilibrium potential (*P < 0.05).

 





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